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Martinez explained the indication for the therapy to the patient and his wife by educating them about ST elevations and the need to treat symptoms quickly in order to provide blood supply to the heart muscle.

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As we discussed in section ii K, the proportion of glycolytic pyruvate being oxidized, and the subsequent intracellular acidosis that follows, may actually be more important with regard to functional outcome, rather than simply increasing glycolytic ATP production.

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Ranolazine at therapeutic levels can inhibit the cardiac late sodium current (INa).

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Psychotherapy, behavioral therapy, and serotonin reuptake inhibitors (SSRIs), which are safe to administer after an acute coronary syndrome, have been shown to reduce mortality and improve recovery from myocardial infarctions (Humphreys, 2011).

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It is possible that sicker patients with PAH, who have more advanced RV dysfunction, would demonstrate even greater benefit from ranolazine than demonstrated in our study; however, additional studies are needed to fully explore the role of ranolazine in high-risk patients with PAH.

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The findings of the OAT study, however, should not be interpreted as applying to all patients experiencing ST-elevation MI, but just to those with a late occluded artery and no- or minimal angina.

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Cardiac overexpression of FACS results in lipid accumulation, cardiac hypertrophy, gradual development of LV dysfunction, and premature death ().

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