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I have been using Iressa for about two month to treat cancer in my lungs. My doctor says this medication prevents cancer cells from growing. Of course, it was only my impression, but I thing Iressa improved my condition because I started feeling myself much better. And after using the medication for about a month my doctor carried out a number of tests and stated that the amount of cancer cells I my lugs decreased. I was really happy to hear that! Not I finally have hope thanks to Iressa!

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The tumor volume ( V t) was represented by an exponential equation describing the actual mean tumor volumes during the 15-d treatment.

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Therapy is identical to that recommended for other non-small cell lung cancer (NSCLC) patients with similar stage disease.

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In this study, gefitinib and erlotinib did not induce apoptosis or necrosis in A549 cells.

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Lung adenocarcinomas from patients who respond to the tyrosine kinase inhibitors gefitinib (Iressa) or erlotinib (Tarceva) usually harbor somatic gain-of-function mutations in exons encoding the kinase domain of the epidermal growth factor receptor (EGFR).

Besides, by using ARMS, we also found 7 samples which harbouring double mutations (2 patients with 19 del and L858R, 1 with L858R and L861Q or S768I, 4 with 19 del and T790M).

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Respiratory Medicine, Graduate School of Medicine, Osaka City University, 1-4-3, Asahi-machi, Abeno-ku, Osaka 545-8585, Japan; Department of Premier Preventive Medicine, Graduate School of Medicine, Osaka City University, 1-4-3, Asahi-machi, Abeno-ku, Osaka 545-8585, Japan.

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Treatment of cell lines with an EGFR inhibitor had no effect on STAT3 levels, but a pan-JAK (see ) inhibitor or an IL6 inhibitor blocked activation of STAT3 and inhibited tumorigenesis.

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Hypercalcemia: Some lung cancers produce substances that remove calcium from bone and release it into the bloodstream, causing a condition called hypercalcemia.

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